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Its expression has been reported in colonic L-cells and appears to be responsible for the glucose-stimulated release of incretins (243, 244)

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This Fc domain serves a dual function: it sterically hinders access to DPP-4, thereby protecting the GLP-1 moiety from enzymatic degradation, and it engages the neonatal Fc receptor (FcRn), facilitating receptor-mediated recycling and preventing lysosomal degradation following cellular uptake

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