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Journal of Diabetes and its Complications, 33 (11), 107414
This burst is a critical mediator of reperfusion injury, overwhelming the compromised antioxidant defense systems and causing widespread cellular damage
From a mechanistic perspective, GLP-1RAs like exenatide notably decreased the percentage of Th17 cells while promoting the proliferation of Tregs by influencing the signaling cascade encompassing phosphatidylinositol 3-kinase (PI3K), protein kinase B (AKT), along with forkhead box O1 (FoxO1) [147]
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