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Combined activation of GLP-1, GIP, and GCG systems theoretically provides complementary mechanisms: GLP-1-mediated insulin sensitization and satiety promotion, GIP-mediated enhancement of insulin secretion and adipose lipid buffering, and glucagon-mediated direct hepatic lipid oxidation and energy expenditure augmentation
A study found that by enhancing FAO and TCA cycles, Sp1-mediated PGE2 up-regulation increased mitochondrial ATP production, leading to glioblastoma (GBM) cell resistance to temozolomide [112]
Am J Physiol Lung Cell Mol Physiol 270 , L1031L1039
Reasons for low levels of initiation and long-term use of AOMs may include reluctance of public health and medical organizations to recognize obesity as a disease, lack of reimbursement, provider inexperience, and misperceptions about the efficacy and safety of available treatments
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