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On the other hand, in HF patients, cardiac energy metabolism is shifted from fatty acid oxidation to glucose, and carnitine insufficiency in the failing myocardium is caused by the leakage of carnitine through damaged cardiomyocyte membranes [4]
(22) showed recently that L-carnitine supplementation promoted improvements in several parameters, including O2 saturation, erythrocyte sedimentation rate (ESR), C-reactive protein (CRP), alkaline phosphatase (ALP) activity, lactate dehydrogenase (LDH), and serum creatine phosphokinase (CPK) when compared to the intervention group
The half-life of acetyl-l-carnitine taken orally is ~4 h [40]
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