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50, 100 and 150 mM CaCl 2

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Acute chemogenetic silencing of NSM neurons suppressed aversion to the mediocre crp and cysE diets, recapitulating the effects of the tph-1 knockout in NSM (Fig

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Mitochondrial complex III deficiency caused by a homozygous UQCRC2 mutation presenting with neonatal-onset recurrent metabolic decompensation

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