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GSH, the most abundant non-protein thiol in cells, acts as a redox buffer to protect cells from oxidative stress and regulate cellular redox signaling, and the thiol group of cysteine in the backbone of GSH is responsible for its antioxidant capacity in maintaining redox homeostasis [36]
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The marked reduction of glycosylated proIGF-1Ea after Tun treatment was not due to general suppression of transcription, as shown by total IGF-1 mRNA quantification (Supplementary Fig
Cc du hiu v triu chng ca tnh trng thiu carnitine th pht bao gm bnh no tng ammonemic (malaise, co git v gim thc do nng amoniac tng), h ng huyt, hketonemia (mc ketone trong mu thp), aciduria dicarboxylic (tng nng axit dicarboxylic trong nc tiu), tng aciduric mu (tha axit uric d trong mu), yu c, myoglobin niu (tha myoglobin trong nc tiu), bnh c tim v t vong t ngt
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