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Other trials have supported the strategy of blocking mTOR/p70S6K as a method to control insulin action and potentially avoid upregulation of IGF-1R signaling by inhibition of mTOR
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The authors inferred that the increased plasma Met concentration in the RPM group, likely supplied the elevated demand in the mammary tissue required to synthesize cytoprotective proteins, such as heat-shock proteins, which are necessary for cellular defense against thermal stress (Pate et al., 2021)
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