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However, previous research in animal models of ASD has shown increased glutamate receptor binding during adolescence instead of childhood, potentially reflecting compensatory mechanisms for earlier synaptic alterations [60]
This is sometimes called metabolic adaptation
Patients with and without anti-tesamorelin IgG antibodies had similar mean reductions in visceral adipose tissue (VAT) and IGF-1 response suggesting that the presence of antibodies did not alter the efficacy of tesamorelin
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