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Retrieved 12 March 2016

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Clinical trials in Alzheimers disease Alzheimers disease is where the 2025 data forced the largest revision, and where the contrast between observational promise and trial outcome is sharpest

The origins of GLP-1s trace back to foundational research in the 1980s, when scientists identified GLP-1 as a hormone critical for regulating insulin secretion and blood glucose levels.[114] Although early findings were promising, the natural form of GLP-1 degraded too rapidly in the body to serve as a viable therapeutic agent.[115] Around the same time, researchers studying the venom of the Gila monster ( Heloderma suspectum ), a lizard native to the southwestern United States, identified a hormone that slows digestion, allowing the animal to survive on just a few meals each year.[116] This research led to the discovery of exendin-4, a molecule that mimics the action of GLP-1 by stimulating insulin secretion