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HIF-1 overexpression or HO-1 agonist treatment suppresses TECs apoptosis by preventing hyperglycemia-induced mitochondrial membrane potential loss and mitochondrial fragmentation to eliminate tubular injury in DKD (108)

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Research published in the Journal of Nutritional Biochemistry (PubMed PMID 22452586) confirmed that CLA supplementation significantly reduced LPL activity in adipose tissue and increased fat oxidation in skeletal muscle in both animal and human study contexts, supporting the mechanistic basis for CLAs anti-fat-storage effect

Scientific Reports, 11(1), 9167